Understanding Bone Loss



Max Stanley Chartrand. Ph.D.
DigiCare® Behavioral Research


Introduction: During their lifetimes, at least half of those over age 50 will be at risk of developing osteoporosis. When we speak of bone loss we are primarily speaking of three diagnostic stages: Osteoarthritis (1-2% loss per annum), Osteopenia (3% per annum), and Osteoporosis (4-5%+ per annum) that are caused almost entirely by diet, hydration, lifestyle, medications, and environ-mental stressors. Rather than attacking under-lying causes in each case, it has become ex-pedient in healthcare to ignore those and go instead for overmedicalized symptoms, false models, and erroneous data in support of a business model that often leaves patients worse off than if they did nothing. The truth is, if one truly desires to restore good bone health they will start by restoring good health first and attacking head-on the avoidable and underlying causes that caused their debilitative state.

Osteoblasts vs Osteoclasts

Human bones are highly vascularized and mineralized tissues that are constantly being shaped and developed by cells called osteoblasts and torn down and resorbed by cells called osteoclasts. Recent research con-firms that throughout one’s lifespan it is osteoblast activity that controls and dictates osteoclast activity as long as the body receives the nutrients it requires to maintain homeostasis. Growing children, for instance, have a far greater abundance of osteoblasts than of osteoclasts. By the time they reach young adulthood (at about age 26 for men, 22 for women) osteo-clasts increase while osteoblasts slow down. Even so, humans of any age can increase osteoblast activity and slow the formation of osteoclasts through weight bearing exercise and other methods. The problem of bone shrinkage and decline in strength presents most often in health states involving:

  • Sedentary Lifestyle, making weight bearing exercise a frontline de-fense against bone loss for everyone.
  • Acidosis (low pH), from a diet that is nutritionally lacking, genetically modified, degerminated, irradiated, laden with toxins & over-processed.
  • Chronic dehydration from too much caffeine and high fructose corn syrup (a GMO) and not enough water that is both ionized and alkalized.
  • Lacking in calcium that is live, ionically charged, as well as phospho-rus, magnesium, boron, and other minerals comprised in human bones. On the other hand, commercially available calcium causes atherosclero-sis, kidney stones, bone spurs, cataracts, and yet MORE bone loss!
  • Taking prescription medications, especially acid reflux meds, NSAIs and steroids. These and more interfere with osteoblast activity and weaken immunology. Osteoporosis meds prevent living bone mass!
  • Unhealed injuries and deterioration of the spine, such as compression fractures (>50% of the US adult population), spinal stenosis, kyphosis, and scoliosis. These cause even more rapid loss of bone mass.
  • Subclinical infections: tooth and gum sepsis, around artificial joints, keratosis obturans, kidney and bladder infections, neuropathies, and osteomyelitis as a result of injuries and/or shock to the bones.
  • Heavy metal accumulations: lead, mercury, cadmium, arsenic, formal-dehyde, cyanide, etc. found in the drinking water, fresh foods, cosmet-ics, paints, fuels, and a host of commonly used products.
  • Lifestyle Substances– Smoking, alcohol, excess coffee, marijuana, opium (including opiate pain killers), diet sodas, caffeine drinks.

Osteoporosis Meds Fraught with Risks

Independent researchers are deeply concerned that poorly designed and self-serving studies and fudged numbers have sold a bill of goods to phy-sicians and consumers on the long-term efficacy of alendronate, etidro-nate, risedronate, bisphosphonate, and zoleronic acid drugs (Fosamax, Boniva, Reclast, etc.). Overprescrib-ing these drugs has brought unfath-omable suffering in the form of: dou-bled cancer risk, ulcers of the esoph-agus, upper GI irritation, fractures of the femur, low blood calcium, skin rash, joint pain, jaw bone decay, increased parathyroid hormone se-cretions, and early death. These drugs simply make the periosteum and teeth brittle, fill the bones with dead bone cells (osteoclasts), and after five years’ use, risk fractures become even more lethal. Filling the bones with dead cells makes bone density tests look good, but the bones need live cells, not dead ones!

Remediation and Treatment

Here are some of the treatment aspects that must be ad-dressed before medication or surgery should be considered:

Diet: Stop microwaving, eat at least 50% of your diet in fresh fruits and vegetables. Avoid GMO high fructose corn syrup and all (processed) artificial sweeteners. Use Extra Virgin Olive Oil, avoid Canola Oil.

Targeted Nutrition: Utilization of Microcrystalline Hydroxyapatite Com-pound (MCHC), etc.; avoid synthetic vitamins/inert minerals.

Hydration: Drink filtered, ionized, alkalized water. Avoid/reduce caffeine.

Heavy Metals: Chelate heavy metals with CardioFlow. Toxins, Medications: As biomarkers improve, with your doctor’s guid-ance, wean off of medications. Avoid tobacco, alcohol, recreational drugs.

Spinal & Accumulated Injuries: All injuries need addressed. As the spine & injuries heal, most neuropathies resolve and bone mass flourish-es. Deep Cold Laser, Far Infra-red, Medical Massage, AromaTouch®, Neuromuscular Retraining, Chiropractics, Dentistry, etc.

Subclinical Infections: Usually found in the jaw and teeth, ear, lungs, artificial joints, feet & intestines. Such are a major cause of inflammation.

Note: Specific treatment programs to accommodate each of the above underlying causes and contributing factors are customized under the SIRCLE® Program to meet the specific needs of each individual.

Resources for Further Study

Brown, S.E. (2014). Osteoporosis risks vs benefits of osteoporosis drugs—spinning the numbers. http://www.betterbones.com/osteoporosis/risks-benefits.aspx.

Chartrand, MS (2013). Introducing Dr. Mitochondria, CG, AZ: DigiCare Behavioral Research.

Clinical Summary (2014). Evaluation of Nutritional Support with Concentrated Microcrystal-line Hydroxyapatite Concentrate. https://nutri-dyn.com/images/LinkedCases/MET1248.pdf

Goldschmidt, V. (2014). Is Sugar Toxic for Your Bones? http://saveourbones.com/is-sugar-toxic-for-your-bones/.

Lee L. (1989). "Health effects of microwave radiation-microwave ovens,"

Lancet Pines, A., Raafat, H., Lynn, A.H., Whttington, J. (1984). Clinical trial of microcrystalline hy-droxyapatite compound in the prevention of osteoporosis due to corticosteroid therapy. Current Medical Research Opinions, 8(10):734-42.

Rust & Kissinger (2008). "BPA leaches from 'safe' products" Journal Sentinel Online.

Villablanca E (December 19, 2007) "Ionizing and non-ionizing radiation: Their difference and possible health consequences.

Yasuda, H. (2012). New roles of osteoblasts in osteoclast differentiation. http://www.wjgnet.com/2218-5836/pdf/v3/i11/175.pdf.